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It is an essential component of the enzyme system that participates in the formation of hemoglobin, carbohydrate metabolism, catecholamine biosynthesis, and also in the formation of cross-links of hair collagen, elastin, and keratin.
Its deficiency causes anemia, neutropenia, and growth disorders in children.
Cu is mainly ingested with food. After absorption, it binds to albumin and ceruloplasmin and is deposited mainly in the liver and, to a lesser extent, in the kidneys. Copper is excreted mainly via the biliary system. Its excretion in the urine is insignificant.
Disruption of MCu homeostasis is associated with a number of diseases, such as Wilson's disease, Menkes syndrome, copper intoxication, and others.
Patient preparation –It is preferable to conduct the study without fasting.
The main part of plasma Cu is bound to ceruloplasmin. Ceruloplasmin is an acute phase protein, therefore, in the acute phase of inflammation, plasma copper concentration increases along with ceruloplasmin: during rheumatoid arthritis, pregnancy, estrogen therapy.
Decreased serum Cu concentration characterizes pathologies with hypoproteinemia (protein deficiency), such as nephrosis, malnutrition, malabsorption. Decreased copper levels also characterize the process of treatment with adrenocorticotropic hormone, glucocorticoids, and valproate.
Blood tests are important for assessing the body's metabolism, blood formation, and nervous system function. Both deficiencies and excesses can cause serious complications — such as Wilson's disease, or Menkes syndrome.
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Testing process
| Purchase a test | Submission of material |
| Results Online | Consult a doctor |
